| [1] CARTHEW R W, RUBIN G M. Seven in absentia, a gene required for specification of R7 cell fate in the Drosophila eye[J]. Cell, 1990, 63(3): 561-577.
[2] DELLA N G, SENIOR P V, BOWTELL D D. Isolation and characterisation of murine homologues of the Drosophila seven in absentia gene (sina)[J]. Development, 1993, 117(4): 1333-1343.
[3] YOSHIBAYASHI H, OKABE H, SATOH S, et al. SIAH1 causes growth arrest and apoptosis in hepatoma cells through beta catenin degradation dependent and independent mechanisms[J]. Oncol Rep, 2007, 17(3): 549-556.
[4] HE H T, FOKAS E, YOU A, et al. Siah1 proteins enhance radiosensitivity of human breast cancer cells[J]. BMC Cancer, 2010, 10: 403.
[5] INOHARA N, DING L, CHEN S, et al. Harakiri, a novel regulator of cell death, encodes a protein that activates apoptosis and interacts selectively with survival promoting proteins Bcl-2 and Bcl-X(L)[J]. EMBO J, 1997, 16(7): 1686-1694.
[6] WEN Y Y, YANG Z Q, SONG M, et al. SIAH1 induced apoptosis by activation of the JNK pathway and inhibited invasion by inactivation of the ERK pathway in breast cancer cells[J]. Cancer Sci, 2010, 101(1): 73-79.
[7] WEN Y Y, YANG Z Q, SONG M, et al. The expression of SIAH1 is downregulated and associated with Bim and apoptosis in human breast cancer tissues and cells[J]. Mol Carcinog, 2010, 49(5): 440-449.
[8] EDGE S B, Byrd D R, Compton C C, et al. AJCC Cancer Staging Manual[M]. 7th ed. New York: Springer, 2010: 347-376.
[9] SOBIN L H, GOAPODAROWIEZ M K, WITTEKIND C. TNM Classification of Malignant Tumors[M]. 7th ed. Hoboken: John Wiley and Sons, 2009. |
