[1]Dellinger RP, Levy MM, Carlet JM, et al. Surviving Sepsis Campaign: international guidelines for management of severe sepsis and septic shock: 2008[J]. Intensive Care Med, 2008, 34(1): 17-60.
[2]Bellomo R. Bench-to-bedside review: lactate and the kidney[J]. Crit Care, 2002, 6(4): 322-326.
[3]Robergs RA, Ghiasvand F, Parker D. Biochemistry of exercise-induced metabolic acidosis[J]. Am J Physiol Regul Integr Comp Physiol, 2004, 287(3): R502-516.
[4]Gore DC, Jahoor F, Hibbert JM, et al. Lactic acidosis during sepsis is related to increased pyruvate production, not deficits in tissue oxygen availability[J]. Ann Surg, 1996, 224(1): 97-102.
[5]Kopterides P, Theodorakopoulou M, Ilias I, et al. Interre-lationship between blood and tissue lactate in a general intensive care unit: a subcutaneous adipose tissue microdialysis study on 162 critically ill patients[J]. J Crit Care, 2012,27(6): 742. e9-18.
[6]Matejovic M, Radermacher P, Fontaine E. Lactate in shock: a high-octane fuel for the heart?[J]. Intensive Care Med, 2007, 33(3): 406-408.
[7]Leverve XM. Energy metabolism in critically ill patients:lactate is a major oxidizable substrate[J]. Curr Opin Clin Nutr Metab Care, 1999, 2(2): 165-169.
[8]Stanley WC, Recchia FA, Lopaschuk GD. Myocardial sub-strate metabolism in the normal and failing heart[J]. Physiol Rev, 2005, 85(3): 1093-1129.
[9]Chatham JC. Lactate—the forgotten fuel![J]. J Physiol,2002, 542(Pt 2): 333.
[10]Nogueira AC, Kawabata V, Biselli P, et al. Changes in plasma free fatty acid levels in septic patients are associated with cardiac damage and reduction in heart rate variability[J]. Shock, 2008, 29(3): 342-348.
[11]Ducrocq N, Kimmoun A, Levy B. Lactate or ScvO2 as an endpoint in resuscitation of shock states?[J]. Minerva Anestesiol, 2013, 79(9): 1049-1058.
[12]Jones AE. Lactate clearance for assessing response to resuscitation in severe Sepsis[J]. acad Emerg Med, 2013, 20(8): 844-847.
[13]James JH, Wagner KR, King JK, et al. Stimulation of both aerobic glycolysis and Na(+)-K(+)-ATPase activity in skele-tal muscle by epinephrine or amylin[J]. Am J Physiol, 1999, 277(1 Pt 1): E176-186.
[14]Bundgaard H, Kjeldsen K, Suarez Krabbe K, et al. Endotox-emia stimulates skeletal muscle Na+-K+-ATPase and raises blood lactate under aerobic conditions in humans[J]. Am J Physiol Heart Circ Physiol, 2003, 284(3): H1028-1034.
[15]Levy B, Mansart A, Montemont C, et al. Myocardial lactate deprivation is associated with decreased cardiovascular performance, decreased myocardial energetics, and early death in endotoxic shock[J]. Intensive Care Med, 2007, 33(3): 495-502.
[16]高峰, 朱永康. 围手术期应激性胰岛素抵抗发生机制研究进展[J]. 温州医学院学报, 2006, 36(2): 185-186.
[17]Trzeciak S, Dellinger RP, Chansky ME, et al. Serum lactate as a predictor of mortality in patients with infection[J]. Intensive Care Med, 2007, 33(6): 970-977.
[18]马光, 洪广亮, 赵光举, 等. 脓毒症患者血浆B型尿钠肽和肌钙蛋白I的变化及意义[J]. 中国中西医结合急救杂志,2014, 21(2): 99-103.
[19]Kang YR, Um SW, Koh WJ, et al. Initial lactate level and mortality in septic shock patients with hepatic dysfunction[J]. Anaesth Intensive Care, 2011, 39(5): 862-867.
[20]Tsai MH, Chen YC, Lien JM, et al. Hemodynamics and metabolic studies on septic shock in patients with acute liver failure J]. J Crit Care, 2008, 23(4): 468-472.
[21]Song YH, Shin TG, Kang MJ, et al. Predicting factors associated with clinical deterioration of sepsis patients with intermediate levels of serum lactate[J]. Shock, 2012, 38(3): 249-254.